These results suggest that PV1 protein is able to block SV40 infectivity at low but not at high viral concentration either by interfering with the infective internalization pathway at the cell surface or at a post internalization step.
Case Report: Suggest activated platelets enhanced coagulation state glomerular expression of PV-1 and endothelial damage may contribute to glomerulonephropathy associated with polycythemia vera.
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