A comparative study of the epiligament of the medial collateral and anterior cruciate ligaments in the human knee: Immunohistochemical analysis of CD 34 alpha-smooth muscle actin and vascular endothelial growth factor in relation to epiligament theory.
Ki67 CD105 and alpha-SMA Expression Supports Biological Distinctness of Oral Squamous Cell Carcinoma Arising in the Background of Oral Submucous Fibrosis.
This study demonstrated that alphasmooth muscle actin could be a predictor for improved postoperative endoscopic and symptomatic outcomes in chronic rhinosinusitis.
Study found that alphaSMA expression is associated with the survival of ER(+) breast cancer patients. Also study observed that wildtype TP53 upregulates alphaSMA expression in tamoxifenresistant breast cancer cells.
Study shows that WT1 functions as a positive regulator of fibroblast activation including fibroproliferation myofibroblast transformation and extracellular matrix production. WT1 binds directly to the promoter DNA sequence of alpha-smooth muscle actin to induce myofibroblast transformation. Genetic lineage tracing identifies WT1 as a key driver of mesothelial-to-myofibroblast and fibroblast-to-myofibroblast transfor...
Ki67 LI CD105 and alpha-SMA expression showed significant differences between normal low-risk oral epithelial dysplasia and high-risk oral epithelial dysplasia. These findings further support that features such as increased basal cell layer hyperplasia abnormal superficial mitosis increased nuclear-cytoplasmic ratio and hyperchromasia could be transformation-relevant dysplastic features.
Adenocarcinomas showed significantly higher staining scores of both VEGF and alphaSMA than squamous cell carcinomas did. In 42 cases of high CD31 score five-year survival rate (87%) of patients with lung cancer showing mature tumor vessels was significantly better than that (69%) of patients with immature tumor vessels
Quantitative real-time PCR and Western blotting revealed these changes to be linked to dose-dependent increases in alphaSMA mRNA and protein expression.
cellular defects due to the ACTA2 mutation in both aortic smooth muscle cells and adventitial fibroblasts may contribute to development of thoracic aortic aneurysms and dissections and proliferative occlusive vascular disease
Cells in high glucose for 7 days showed a significant decrease in mRNA expression of CD31 and VE-cadherin and a significant increase in that of alpha-SMA and collagen I.
Alpha-SMA overexpression induced YAP translocation to the nucleus and reduced the high clonogenicity and adipogenic potential of alpha-SMA-negative MSCs.
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