These findings for the first time revealed that miR-598 as a tumor suppressor negatively regulate DERL1 and Epithelial-Mesenchymal Transition to suppress the invasion and migration in Non-Small Cell Lung Cancer (NSCLC) thereby putatively serving as a novel therapeutic target for NSCLC clinical treatment.
association of the degradation factor HRD1 with the translocon and the rerouting factor Derlin-1 may be necessary for the smooth and effective clearance of ERpQC substrates
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