The data identify FADD as a novel component of the noncanonical TLR4/IFN-beta signaling pathway and demonstrate that both Fas and its adaptor FADD can differentially regulate the production of LPS-induced proinflammatory cytokines and type I interferons.
REVIEW: recent knowledge of the biological roles of FADD a pleiotropic molecule having multiple partners and its impact in cancer innate immunity and inflammation
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