These results define a model for the study of inflammatory lymphangiogenesis in the thyroid and implicate lymphotoxin beta receptor signaling in this process.
These observations suggest that LTBR which belongs to the tumor necrosis factor receptor superfamily of proteins may contribute to human carcinogenesis.
Embryonic LTbetaR is expressed from E16.5 onward in respiratory salivary bronchial and gastric epithelium which may be consistent with early communication events between lymphoid elements and epithelial specialization in emerging mucosal lymph nodes.
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