study reports an alternately-spliced form of Smad7 Smad7Delta that is induced by TGF-beta and CLIC4 is a dominant inhibitor of Smad7 and enhances TGF-beta signaling
describe a conditional system for expressing Smad7 a potent inhibitor of transforming growth factor beta 1 signaling to identify distinct roles for this pathway in adult and embryonic beta cells
relative Smad7 deficiency observed in a scleroderma model may account for sustained activation of transforming growth factor-beta/Smad signaling in lesional tissues.
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