Paternal stress alters synaptic density and expression of GAP-43 GRIN1 M1 and SYP genes in the hippocampus and cortex of offspring of stress-induced male rats.
Study shows that functional ectopic neural lobe increases GAP-43 expression via PI3K/AKT pathways to alleviate central diabetes insipidus after pituitary stalk lesion in rats.
Data show that growth-associated protein 43 (GAP43) gene-modified bone marrow-derived mesenchymal stem cells (BMSCs) transplantation can increase survival photoreceptor cells and delay retinal degeneration
Results show that repeated contingent but not non-contingent cocaine self-administration promotes an increase of nELAV expression immediately and 7 days after the last cocaine self-administration session with a transient disappearance observed 24 h later. The evidence that the ubiquitous ELAV/HuR is not changed calls for a specific involvement of nELAV in these molecular mechanisms.
Data show that the paw skin and back skin revealed a significantly higher number of nerve fibres expressing growth-associated protein 43 (GAP-43) at both the acute and chronic stages of antigen-induced arthritis (AIA).
The manifestation of clinical signs of experimental autoimmune encephalomyelitis has been found to be in correlation with the levels of GAP-43 proteolysis.
Results show that PKC-dependent phosphorylation of GAP43 plays a critical role in regulating postsynaptic gephyrin aggregation in developing GABAergic synapses.
There was no significant difference in immunohistochemical staining for transforming growth factor beta 3 between the control injured and obese injured groups
Progesterone promotes neuroprotection following traumatic brain injury by inhibiting the expression of Nogo-A and GFAP and increasing GAP-43 expression.
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