these findings indicated that plectin protects podocytes from ADR-induced apoptosis and F-actin cytoskeletal disruption by inhibiting integrin alpha6beta4/FAK/p38 pathway activation and that plectin may be a therapeutic target for podocyte injury-related glomerular diseases.
propose that the caspase 8-mediated degradation of plectin represents a critical biochemical event during CCK-induced secretory blockade and pancreatic acinar cell injury
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