Two mechanistically distinct PIN3 polarization events redirect auxin fluxes at different time points of the gravity response: first gravity-mediated redirection of PIN3-mediated auxin flow toward the lower hypocotyl side where auxin gradually accumulates and promotes growth and later PIN3 polarization to the opposite cell side depleting this auxin maximum to end the bending.
FLP and MYB88 expression specifically determines the temporal-spatial patterns of PIN3 and PIN7 transcription that are closely associated with their preferential functions during root responses to gravity.
ARF7 and FLP transcription factors jointly form a coherent feed-forward motif that mediates the auxin-responsive PIN3 transcription in planta to steer the early steps of lateral root formation.
Data show that phototropic hypocotyl bending is strongly dependent on the activity of D6PKs and the PIN-FORMED (PIN) auxin efflux facilitators PIN3 PIN4 and PIN7.
The loss of PIN3 causes a dramatic delay in the onset of Lateral root initiation (LRI) after founder cell (FC) are specified which can be fully recovered by the expression of PIN3 exclusively in the endodermis.
Mutation of phosphorylation sites caused defects in PIN3-mediated biological processes such as auxin efflux activity auxin maxima formation root growth and root gravitropism.
PIP5K2 is involved in regulating lateral root formation and root gravity response and reveal a critical role of PIP5K2/PtdIns(4 5)P(2) in root development through regulation of PIN proteins providing direct evidence of crosstalk between the phosphatidylinositol signaling pathway and auxin response and new insights into the control of polar auxin transport
suggest that 1-aminocyclopropane-1-carboxylic acid treatment increased PIN3 and PIN7 expression resulting in elevated auxin transport which prevented the localized accumulation of auxin needed to drive lateral root formation
Data suggest a role of gibberellic acid (GA) in Arabidopsis growth responses and for a GA-dependent modulation of PIN1 -2 and -3 turnover that may be causative for these differential growth responses.
Data show that light polarizes the cellular localization of the auxin efflux carrier PIN3 in hypocotyl endodermis cells resulting in changes in auxin distribution and differential growth.
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