PHOSPHATE STARVATION RESPONSE1 (PHR1) interacts with JASMONATE ZIM-DOMAIN (JAZ) and MYC2 to modulate phosphate deficiency-induced jasmonate signaling in Arabidopsis.
MBW complex consisting of three kinds of regulatory proteins MYB and basic helix-loop-helix (bHLH) transcription factors and a WD40 repeat protein TRANSPARENT TESTA GLABRA1 (TTG1) acts in concert to promote trichome formation and flavonoid accumulation in Arabidopsis thaliana. TTG1 functions as an essential activator in these two biological processes.
Mediator subunit MED25 physically recruits LUH to MYC2 target promoters that then links MYC2 with HAC1-dependent acetylation of Lys-9 of histone H3 (H3K9ac) to activate JAZ2 and LOX2 Moreover LUH promotes hormone-dependent enhancement of protein interactions between MYC2 and its coactivators MED25 and HAC1.
The receptor kinase FERONIA (FER) functions to inhibit JA and COR signaling by phosphorylating and destabilizing MYC2 thereby positively regulating immunity. Conversely the peptide ligand RALF23 acts through FER to stabilize MYC2 and elevate JA signaling negatively contributing to plant immunity.
The authors report a link between abscisic acid (ABA) and jasmonic Acid (JA) signaling through a direct interaction of the ABA receptor PYL6 (RCAR9) with the basic helix-loop-helix transcription factor MYC2. PYL6 and MYC2 interact in yeast two hybrid assays and the interaction is enhanced in the presence of ABA.
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